Showing 1 - 8 results of 8 for search 'P.U. Cameron', query time: 0.04s Refine Results
  1. 1
    Co-culture of T-cells with dendritic cells facilitates HIV latency in proliferating CD4+ T-cells: implications for the establishment and reversal of latency

    Co-culture of T-cells with dendritic cells facilitates HIV latency in proliferating CD4+ T-cells: implications for the establishment and reversal of latency by N. Kumar, R.M. van der Sluis, T. Mota, V.A. Evans, S.R. Lewin, P.U. Cameron

    Published 2015
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  2. 2
    31 High prevalence of CXCR4-using virus and low clonal expansion in infected naïve and central memory CD4+ T cells in individuals on ART

    31 High prevalence of CXCR4-using virus and low clonal expansion in infected naïve and central memory CD4+ T cells in individuals on ART by M. Roche, C. Tumpach, J. Symons, P.U. Cameron, M.J. Churchill, S.G. Deeks, P.R. Gorry, S.R. Lewin

    Published 2017
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  3. 3
    8 IFNα activates latent HIV-1 in non-proliferating latently infected T-cells

    8 IFNα activates latent HIV-1 in non-proliferating latently infected T-cells by R.M. Sluis, van der, N.A. Kumar, V.A. Evans, A.N. Harman, T. Mota, S. Tennakoon, P. Herzog, S.R. Lewin, P.U. Cameron

    Published 2016
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  4. 4
    32 Plasmacytoid DCs control HIV latency in resting T cells by type I IFNα

    32 Plasmacytoid DCs control HIV latency in resting T cells by type I IFNα by R.M. Van der Sluis, T. Mota, N.A. Kumar, V.A. Evans, A.N. Harman, S. Tennakoon, S.G. Deeks, P. Herzog, S.R. Lewin, P.U. Cameron

    Published 2017
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  5. 5
    OA3-3 Anti-PD-1 disrupts HIV latency in non-proliferating but not in proliferating T cells

    OA3-3 Anti-PD-1 disrupts HIV latency in non-proliferating but not in proliferating T cells by R.M. Van der Sluis, N.A. Kumar, V.A. Evans, A.I. Dantanarayana, R.P. Sékaly, R. Fromentin, N. Chomont, P.U. Cameron, S.R. Lewin

    Published 2017
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  6. 6
    Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART

    Clonal integration site expansion of infected cells is a main contributor of HIV persistence in more differentiated T cell subsets during suppressive ART by J. Symons, C. Bacchus-Souffan, A. Chopra, S. Leary, D. Cameron, P.U. Cameron, R. Hoh, H. Ahn, S.G. Deeks, J.M. McCune, S. Mallal, P.W. Hunt, S.R. Lewin

    Published 2019
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  7. 7
    23 Understanding the effects of latency reversing agents on HIV RNA splicing: implications for latency reversal

    23 Understanding the effects of latency reversing agents on HIV RNA splicing: implications for latency reversal by T.M. Mota, G. Khoury, J.C. Jacobson, H.K. Lu, F. Wightman, d.e.r. Sluis, J.L. Anderson, P.U. Cameron, S.R. Lewin, D.F.J. Purcell, R.M. Van

    Published 2016
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  8. 8
    The CCR5-agonist maraviroc reverses HIV latency, results from ex vivo studies and a randomized placebo controlled clinical trial

    The CCR5-agonist maraviroc reverses HIV latency, results from ex vivo studies and a randomized placebo controlled clinical trial by J. Symons, S.F.L. van Lelyveld, W. de Spiegelaere, A.M.J. Wensing, J. Drylewicz, A.I.M. Hoepelman, P.U. Cameron, H. Lu, T. Mota, A.I. Dantanarayana, L. Vandekerckhove, S.R. Lewin, K. Tesselaar, M. Nijhuis

    Published 2017
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