New Perspective in Atrial Fibrillation

In spite of the large volume of associated research, the pathophysiological mechanisms involved in atrial fibrillation (AF) onset and recurrence remain uncertain. This may explain why the performances of thromboembolic and bleeding prediction scores in AF patients are limited. In the past few years,...

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Bibliographic Details
Other Authors: Guenancia, Charles (Editor)
Format: Electronic Book Chapter
Language:English
Published: Basel, Switzerland MDPI - Multidisciplinary Digital Publishing Institute 2021
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520 |a In spite of the large volume of associated research, the pathophysiological mechanisms involved in atrial fibrillation (AF) onset and recurrence remain uncertain. This may explain why the performances of thromboembolic and bleeding prediction scores in AF patients are limited. In the past few years, the concept of atrial cardiopathy has emerged as a promising lead to connect AF to stroke, heart failure, and inflammatory processes: indeed, all of the mechanisms associated with atrial remodeling and the development of atrial cardiopathy are also likely to promote the development of AF. This recent concept of atrial cardiopathy suggests that the real trigger of stroke may be an abnormal atrial substrate rather than atrial rhythm itself. In this setting, AF could be seen as a symptom of atrial cardiopathy rather than a risk factor of stroke. In the absence of validated clinical markers of atrial cardiopathy, the search for the mechanism of AF remains the cornerstone of cardioembolic stroke prevention for now.The aim of this Special Issue is to gather basic research as well as pathophysiological and epidemiological papers focused on the relationship between atrial substrates and atrial fibrillation onset, recurrence, and outcomes. 
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653 |a postoperative atrial fibrillation 
653 |a biomarkers 
653 |a coronary artery bypass grafting 
653 |a miRNA 
653 |a circRNA 
653 |a mtDNA 
653 |a SNPs 
653 |a atrial fibrillation 
653 |a acute myocardial infarction 
653 |a heart rate variability 
653 |a autonomic nervous system 
653 |a bleeding risk 
653 |a age 
653 |a left atrial appendage closure 
653 |a coronary artery disease 
653 |a prognosis 
653 |a anticoagulation 
653 |a antiplatelet therapy 
653 |a hypertension 
653 |a elderly 
653 |a prevention 
653 |a ganglionated plexi 
653 |a ablation 
653 |a dementia 
653 |a early-onset 
653 |a n/a 
653 |a ischemic stroke 
653 |a risk factor 
653 |a new oral anticoagulants 
653 |a mitochondria 
653 |a cardiac remodeling 
653 |a pharmacotherapy 
653 |a electroporation 
653 |a pulsed field ablation 
653 |a cardiac 
653 |a heart 
653 |a arrhythmia 
653 |a NOAC 
653 |a VKA 
653 |a statin 
653 |a outcome 
653 |a mortality 
653 |a paroxysmal atrial fibrillation 
653 |a catheter ablation 
653 |a epicardial adipose tissue 
653 |a persistent atrial fibrillation 
653 |a posterior wall 
653 |a hybrid ablation 
653 |a convergent ablation 
653 |a atrial tachycardia 
653 |a mechanism 
653 |a spatiotemporal dispersion 
653 |a left atrial appendage occlusion 
653 |a BNP 
653 |a atrial remodeling 
653 |a atrial cardiopathy 
653 |a cardiac ablation 
653 |a irreversible electroporation 
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