Acute nitric oxide synthase inhibition and endothelin-1-dependent arterial pressure elevation
Key evidence that endogenous nitric oxide (NO) inhibits the continuous, endothelin (ET)-1-mediated drive to elevate arterial pressure includes demonstrations that ET-1 mediates a significant component of the pressure elevated by acute exposure to NO synthase (NOS) inhibitors. This review examines th...
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Frontiers Media S.A.,
2014-04-01T00:00:00Z.
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