Exogenous S1P Exposure Potentiates Ischemic Stroke Damage That Is Reduced Possibly by Inhibiting S1P Receptor Signaling
Initial and recurrent stroke produces central nervous system (CNS) damage, involving neuroinflammation. Receptor-mediated S1P signaling can influence neuroinflammation and has been implicated in cerebral ischemia through effects on the immune system. However, S1P-mediated events also occur within th...
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Main Authors: | Eunjung Moon (Author), Jeong Eun Han (Author), Sejin Jeon (Author), Jong Hoon Ryu (Author), Ji Woong Choi (Author), Jerold Chun (Author) |
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Format: | Book |
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Hindawi Limited,
2015-01-01T00:00:00Z.
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Online Access: | Connect to this object online. |
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