Long-Lasting Effect of Infant Rats Endotoxemia on Heat Shock Protein 60 in the Pancreatic Acinar Cells: Involvement of Toll-Like Receptor 4
Introduction. Lipopolysaccharide endotoxin (LPS) is responsible for septic shock and multiorgan failure, but pretreatment of rats with low doses of LPS reduced pancreatic acute damage. Aim. We investigated the effects of the endotoxemia induced in the early period of life on Toll-like receptor 4 (TL...
Saved in:
Main Authors: | Joanna Bonior (Author), Jolanta Jaworek (Author), Michalina Kot (Author), Stanisław J. Konturek (Author), Piotr Pierzchalski (Author) |
---|---|
Format: | Book |
Published: |
Hindawi Limited,
2012-01-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Protective Effect of Melatonin on Acute Pancreatitis
by: Jolanta Jaworek, et al.
Published: (2012) -
Nutrition and Supplementation Considerations to Limit Endotoxemia When Exercising in the Heat
by: Joshua H. Guy, et al.
Published: (2018) -
Nanomolar resveratrol reduces early alterations of pancreatitis and pancreatic cancer in pancreatic acinar cells
by: Thiago M.C. Pereira, et al.
Published: (2022) -
Immune Sensitization to the 60 kD Heat Shock Protein and Pregnancy Outcome
by: A. Neuer, et al.
Published: (1997) -
Mixed Acinar-Endocrine Carcinoma (MAEC) Arising in Duodenal Pancreatic Heterotopia
by: Phoenix D. Bell, et al.
Published: (2019)