Neutrophils releasing IL-17A into NETs are essential to plasma cell differentiation in inflamed tissue dependent on IL-1R
Interleukin (IL) 17A in chronic inflammation is also produced by innate immune cells as neutrophils. Mice with chronic humoral response induced by venom of Thalassophryne nattereri (VTn) proved to be a good tool for evaluating the impact of IL-17A on the development of long-lived plasma cells in the...
Saved in:
Main Authors: | Lidiane Zito Grund (Author), Ivan Novaski (Author), Valerie F. Quesniaux (Author), Bernhard Ryffel (Author), Monica Lopes-Ferreira (Author), Carla Lima (Author) |
---|---|
Format: | Book |
Published: |
Taylor & Francis Group,
2017-02-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
TLR2 Elicits IL-17-Mediated RANKL Expression, IL-17, and OPG Production in Neutrophils from Arthritic Mice
by: Viktoriya Milanova, et al.
Published: (2014) -
IL-17 and IL-22 in Cerebrospinal Fluid and Plasma Are Elevated in Guillain-Barré Syndrome
by: Shujuan Li, et al.
Published: (2012) -
Does IL-17 Respond to the Disordered Lung Microbiome and Contribute to the Neutrophilic Phenotype in Asthma?
by: Xu Yang, et al.
Published: (2016) -
Nrf2 Deficiency Accelerates IL-17-Dependent Neutrophilic Airway Inflammation in Asthmatic Mice
by: Kenya Kuramoto, et al.
Published: (2024) -
Clinical Significance of IL-23 Regulating IL-17A and/or IL-17F Positive Th17 Cells in Chronic Periodontitis
by: Zhenhua Luo, et al.
Published: (2014)