Suppressing the Substance P-NK1R Signalling Protects Mice against Sepsis-Associated Acute Inflammatory Injury and Ferroptosis in the Liver and Lungs
Substance P (SP), encoded by the <i>TAC1/Tac1</i> gene, acts as a significant mediator in dysregulated systemic inflammatory response and associated organ injury in sepsis by activating the neurokinin-1 receptor (NK1R). This study investigated the impact of SP-NK1R signaling on ferroptos...
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| Main Authors: | , , |
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| Format: | Book |
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MDPI AG,
2024-02-01T00:00:00Z.
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