Tissue Kallikrein supplementation in ischemic phase protects the neurovascular unit and attenuates reperfusion-induced injury in ischemic stroke
Tissue kallikrein (TK) has emerged as a potential neuroprotective agent in ischemic stroke (IS), yet the optimal timing and mechanisms of TK therapy remain unclear. Here, we established a causal link between lower baseline TK levels and an increased risk of stroke through a retrospective, multicente...
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| Main Authors: | , , , , |
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| Format: | Book |
| Published: |
Elsevier,
2024-11-01T00:00:00Z.
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| Online Access: | Connect to this object online. |
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