Cathelicidin Modulates Vascular Smooth Muscle Cell Phenotypic Switching through ROS/IL-6 Pathway
Vascular smooth muscle cells (VSMC) are stromal cells of the blood vessels and their differentiation is thought to be essential during atherosclerosis. Cathelicidin-related antimicrobial peptides (CRAMP) are suggested to play a role in the development of atherosclerosis. Even so, the relationship of...
Saved in:
Main Authors: | Xiaoliang Dong (Author), Di Wu (Author), Yihan Zhang (Author), Lingling Jia (Author), Xiaohua Pan (Author), Jia Sun (Author), Li-Long Pan (Author) |
---|---|
Format: | Book |
Published: |
MDPI AG,
2020-06-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Induction of ferroptosis promotes vascular smooth muscle cell phenotypic switching and aggravates neointimal hyperplasia in mice
by: Shunchi Zhang, et al.
Published: (2022) -
circ_TGFBR2 Inhibits Vascular Smooth Muscle Cells Phenotypic Switch and Suppresses Aortic Dissection Progression by Sponging miR-29a
by: Xu Z, et al.
Published: (2021) -
Phenotypic Modulation of Macrophages and Vascular Smooth Muscle Cells in Atherosclerosis-Nitro-Redox Interconnections
by: Justine Bonetti, et al.
Published: (2021) -
Global trends and Frontier topics about vascular smooth muscle cells phenotype switch: A bibliometric analysis from 1999 to 2021
by: Ying Han, et al.
Published: (2022) -
Chloroquine Attenuates Asthma Development by Restoring Airway Smooth Muscle Cell Phenotype Via the ROS-AKT Pathway
by: Yan Ren, et al.
Published: (2022)