Lack of the Nlrp3 Inflammasome Improves Mice Recovery Following Traumatic Brain Injury

• Үндсэн зохиолчид: Natasha Irrera (Зохиогч), Gabriele Pizzino (Зохиогч), Margherita Calò (Зохиогч), Giovanni Pallio (Зохиогч), Federica Mannino (Зохиогч), Fausto Famà (Зохиогч), Vincenzo Arcoraci (Зохиогч), Vincenzo Fodale (Зохиогч), Antonio David (Зохиогч), Cosentino Francesca (Зохиогч), Letteria Minutoli (Зохиогч), Emanuela Mazzon (Зохиогч), Placido Bramanti (Зохиогч), Francesco Squadrito (Зохиогч), Domenica Altavilla (Зохиогч), Alessandra Bitto (Зохиогч)
• Формат: Ном
• Хэвлэсэн: Frontiers Media S.A., 2017-07-01T00:00:00Z.
• Нөхцлүүд: article
• Онлайн хандалт: Connect to this object online.

Treatment for traumatic brain injury (TBI) remains elusive despite compelling evidence from animal models for a variety of therapeutic targets. The activation of the NLRP3 (Nucleotide-binding oligomerization domain-like receptor family pyrin domain-containing 3) inflammasome has been proposed as key...