Autophagy Improves Inflammatory Response in Sepsis Accompanied by Changes in Gut Microbiota
Conclusion: Autophagy is able to improve inflammation and may directly or indirectly regulate the microbiota of septic rats. Autophagy may be an important target for future clinical interventions in the treatment of sepsis.
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Format: | Book |
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Hindawi Limited,
2024-01-01T00:00:00Z.
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001 | doaj_c9f5f732c0434d4e9d215e3419bf1b98 | ||
042 | |a dc | ||
100 | 1 | 0 | |a La Wang |e author |
700 | 1 | 0 | |a WenJia Wang |e author |
700 | 1 | 0 | |a GuiTong Jiang |e author |
700 | 1 | 0 | |a ZunLi Ke |e author |
700 | 1 | 0 | |a RuiXi Luo |e author |
700 | 1 | 0 | |a WeiYi Tian |e author |
245 | 0 | 0 | |a Autophagy Improves Inflammatory Response in Sepsis Accompanied by Changes in Gut Microbiota |
260 | |b Hindawi Limited, |c 2024-01-01T00:00:00Z. | ||
500 | |a 1466-1861 | ||
500 | |a 10.1155/2024/9550301 | ||
520 | |a Conclusion: Autophagy is able to improve inflammation and may directly or indirectly regulate the microbiota of septic rats. Autophagy may be an important target for future clinical interventions in the treatment of sepsis. | ||
546 | |a EN | ||
690 | |a Pathology | ||
690 | |a RB1-214 | ||
655 | 7 | |a article |2 local | |
786 | 0 | |n Mediators of Inflammation, Vol 2024 (2024) | |
787 | 0 | |n http://dx.doi.org/10.1155/2024/9550301 | |
787 | 0 | |n https://doaj.org/toc/1466-1861 | |
856 | 4 | 1 | |u https://doaj.org/article/c9f5f732c0434d4e9d215e3419bf1b98 |z Connect to this object online. |