H3K4me3 mediates uterine leiomyoma pathogenesis via neuronal processes, synapsis components, proliferation, and Wnt/β-catenin and TGF-β pathways

• Asıl Yazarlar: María Cristina Carbajo-García (Yazar), Elena Juarez-Barber (Yazar), Marina Segura-Benítez (Yazar), Amparo Faus (Yazar), Alexandra Trelis (Yazar), Javier Monleón (Yazar), Greta Carmona-Antoñanzas (Yazar), Antonio Pellicer (Yazar), James M. Flanagan (Yazar), Hortensia Ferrero (Yazar)
• Materyal Türü: Kitap
• Baskı/Yayın Bilgisi: BMC, 2023-01-01T00:00:00Z.
• Konular: article
• Online Erişim: Connect to this object online.

Abstract Background Uterine leiomyomas (UL) are the most common benign tumor in women of reproductive age. Their pathology remains unclear, which hampers the development of safe and effective treatments. Raising evidence suggests epigenetics as a main mechanism involved in tumor development. Histone...