Muscle-specific lack of Gfpt1 triggers ER stress to alleviate misfolded protein accumulation

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Bibliographic Details
Main Authors: Ruchen Zhang (Author), Paniz Farshadyeganeh (Author), Bisei Ohkawara (Author), Kazuki Nakajima (Author), Jun-ichi Takeda (Author), Mikako Ito (Author), Shaochuan Zhang (Author), Yuki Miyasaka (Author), Tamio Ohno (Author), Madoka Mori-Yoshimura (Author), Akio Masuda (Author), Kinji Ohno (Author)
Format: Book
Published: The Company of Biologists, 2024-08-01T00:00:00Z.
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001 doaj_dd1d6d2ad7d74c9db299f98aace79c6f
042 |a dc 
100 1 0 |a Ruchen Zhang  |e author 
700 1 0 |a Paniz Farshadyeganeh  |e author 
700 1 0 |a Bisei Ohkawara  |e author 
700 1 0 |a Kazuki Nakajima  |e author 
700 1 0 |a Jun-ichi Takeda  |e author 
700 1 0 |a Mikako Ito  |e author 
700 1 0 |a Shaochuan Zhang  |e author 
700 1 0 |a Yuki Miyasaka  |e author 
700 1 0 |a Tamio Ohno  |e author 
700 1 0 |a Madoka Mori-Yoshimura  |e author 
700 1 0 |a Akio Masuda  |e author 
700 1 0 |a Kinji Ohno  |e author 
245 0 0 |a Muscle-specific lack of Gfpt1 triggers ER stress to alleviate misfolded protein accumulation 
260 |b The Company of Biologists,   |c 2024-08-01T00:00:00Z. 
500 |a 1754-8403 
500 |a 1754-8411 
500 |a 10.1242/dmm.050768 
546 |a EN 
690 |a glutamine fructose-6-phosphate transaminase 1 (gfpt1) 
690 |a congenital myasthenic syndrome (cms) 
690 |a neuromuscular junction (nmj) 
690 |a hexosamine biosynthesis pathway (hbp) 
690 |a endoplasmic reticulum (er) stress 
690 |a unfolded protein response (upr) 
690 |a Medicine 
690 |a R 
690 |a Pathology 
690 |a RB1-214 
655 7 |a article  |2 local 
786 0 |n Disease Models & Mechanisms, Vol 17, Iss 8 (2024) 
787 0 |n http://dmm.biologists.org/content/17/8/dmm050768 
787 0 |n https://doaj.org/toc/1754-8403 
787 0 |n https://doaj.org/toc/1754-8411 
856 4 1 |u https://doaj.org/article/dd1d6d2ad7d74c9db299f98aace79c6f  |z Connect to this object online.