The gliadin-CFTR connection: new perspectives for the treatment of celiac disease
Abstract Familial loss-of-function mutations of the gene coding for the cystic fibrosis transmembrane conductance regulator (CFTR) channel protein cause cystic fibrosis (CF), the most frequent inherited life-threatening disease in the Caucasian population. A recent study indicates that the gluten/gl...
Saved in:
Main Authors: | Luigi Maiuri (Author), Valeria R. Villella (Author), Valeria Raia (Author), Guido Kroemer (Author) |
---|---|
Format: | Book |
Published: |
BMC,
2019-03-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Targeting the intracellular environment in cystic fibrosis: restoring autophagy as a novel strategy to circumvent the CFTR defect
by: Valeria Rachela Villella, et al.
Published: (2013) -
Performance of deamidated gliadin peptide antibodies as first screening for celiac disease in the general pediatric population
by: Abdulrahman Al-Hussaini, et al.
Published: (2023) -
Relation of ?-Amylase Activity with Glucose and Anti-Gliadin IgA and IgG in Sera of Patients with Celiac Disease
by: Sura A. Al-Emami, et al.
Published: (2017) -
Relation of ?-Amylase Activity with Glucose and Anti-Gliadin IgA and IgG in Sera of Patients with Celiac Disease
by: Sura A. Al-Emami, et al.
Published: (2017) -
Relation of ?-Amylase Activity with Glucose and Anti-Gliadin IgA and IgG in Sera of Patients with Celiac Disease
by: Sura A. Al-Emami, et al.
Published: (2017)