Nature of Human Gut Microbiome: How do they play in Cardiovascular Disease?
<p>Cardiovascular disease is the number one killer of death around the world. Most of the cardiovascular</p><p>diseases are caused by sedentary life style, bad eating habit, tobacco smoking, high alcohol intake,</p><p>dyslipidemia and genetic factors .Recently the idea...
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Journal of Cardiovascular Medicine and Cardiology - Peertechz Publications,
2018-11-05.
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LEADER | 00000 am a22000003u 4500 | ||
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001 | peertech__10_17352_2455-2976_000074 | ||
042 | |a dc | ||
100 | 1 | 0 | |a Han Naung Tun |e author |
245 | 0 | 0 | |a Nature of Human Gut Microbiome: How do they play in Cardiovascular Disease? |
260 | |b Journal of Cardiovascular Medicine and Cardiology - Peertechz Publications, |c 2018-11-05. | ||
520 | |a <p>Cardiovascular disease is the number one killer of death around the world. Most of the cardiovascular</p><p>diseases are caused by sedentary life style, bad eating habit, tobacco smoking, high alcohol intake,</p><p>dyslipidemia and genetic factors .Recently the idea of human microbiome science has emerged</p><p>in diseases pathogenesis .The human gut is a house of trillions of microbial fl oral. Since a couple of</p><p>decades ago, there has been interesting insights into the human gut microbiota and have highlighted</p><p>its increasingly association to cardiovascular (CV) and metabolic diseases. Trimethylamine N-oxide</p><p>(TMAO), which is a metabolic product from gut microbiota plays a central role in cardiovascular disease</p><p>pathogenesis. Nature of microbial inhabitants within the host has been noticed by the numbers of</p><p>scientists and researchers to understand more about the hidden mechanism of diseases pathogenesis</p><p>including cardiovascular disease, metabolic and autoimmune diseases and it has become a good hope to</p><p>develop new drug designs to prevent metabolic and cardiovascular disease in near future.</p> | ||
540 | |a Copyright © Han Naung Tun et al. | ||
546 | |a en | ||
655 | 7 | |a Review Article |2 local | |
856 | 4 | 1 | |u https://doi.org/10.17352/2455-2976.000074 |z Connect to this object online. |