Nutrigenomics Coupling with other OMICS Platform Enhance Personalized Health Care in Metabolic Disorders

<p>Diabetes is a multifactorial of diseases characterized by high blood glucose levels which occur as a result in the body's inability to produce and/or use insulin. Both type 1 and 2 diabetes are thought to be complex disease which developed by the influence of many susceptibility and pr...

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Bibliographic Details
Main Authors: Sylvester Ndimele (Author), Miranda L. Carpenter (Author), Jae-Hyeon Cho (Author), Hong Duck Kim (Author)
Format: Book
Published: Global Journal of Obesity, Diabetes and Metabolic Syndrome - Peertechz Publications, 2017-02-03.
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001 peertech__10_17352_2455-8583_000016
042 |a dc 
100 1 0 |a Sylvester Ndimele  |e author 
700 1 0 |a  Miranda L. Carpenter  |e author 
700 1 0 |a  Jae-Hyeon Cho  |e author 
700 1 0 |a Hong Duck Kim  |e author 
245 0 0 |a Nutrigenomics Coupling with other OMICS Platform Enhance Personalized Health Care in Metabolic Disorders 
260 |b Global Journal of Obesity, Diabetes and Metabolic Syndrome - Peertechz Publications,   |c 2017-02-03. 
520 |a <p>Diabetes is a multifactorial of diseases characterized by high blood glucose levels which occur as a result in the body's inability to produce and/or use insulin. Both type 1 and 2 diabetes are thought to be complex disease which developed by the influence of many susceptibility and protective genes, in relation with negative and positive environmental factors. Though type 1 diabetes is distinguished by common beta-cell loss which is mediated by an autoimmune process to extent that all patients with overt type 1 diabetes will essentially need insulin. Multiple genetic factors have been connected to type 1 diabetes which can define individualized plan for type 1 prevention. This review has focused on type 2 diabetes (T2D) which has become more and more a challenging health burden as a result of its degree of morbidity, mortality and heightened prevalence worldwide.</p> 
540 |a Copyright © Sylvester Ndimele et al. 
546 |a en 
655 7 |a Short Communication  |2 local 
856 4 1 |u https://doi.org/10.17352/2455-8583.000016  |z Connect to this object online.