Kidney Inflammation, Injury and Regeneration

• Hoofdauteur: Koch, Benjamin (auth)
• Andere auteurs: Geiger, Helmut (auth), Baer, Patrick C. (auth)
• Formaat: Elektronisch Hoofdstuk
• Taal: Engels
• Gepubliceerd in: MDPI - Multidisciplinary Digital Publishing Institute 2020
• Onderwerpen: Medicine; microRNAs; n/a; transcription; ischemia/reperfusion injury; DSS-colitis; kidney inflammation; therapeutics targets; CXCL13; glomerulus; interleukin-6; rhabdomyolysis; IgA nephropathy; CREB Regulated Transcriptional Coactivators (CRTC); slit diaphragm; injury; xanthine oxidase; Salt Inducible Kinase (SIK); acute and chronic kidney disease; therapeutic target; KIT-IgA score; G-protein-coupled bile acid receptor (TGR5); lysophosphatidic acid; glomerular injury; IL-18; mesenchymal stem cells; Taiwan; acute kidney injury; renal ischemia-reperfusion; long non-coding RNA; fibrosis; acute kidney failure; diabetic kidney diseases; chronic kidney disease; lncRNA; LPS-binding protein; endotoxemia-induced oliguric kidney injury; dapagliflozin; cPLA2 and COX-2; NLRP3 inflammasome; CmklR1; haem; chronic kidney injury; omega-3 fatty acid; noninvasive; inflammation; regulated necrosis; GLP-1 receptor agonists; miRNA; AKI; SGLT2 inhibitors; diabetic kidney disease; extracellular vesicles; podocin; type IV collagen; epithelial cells; nephrin; 2-kidney-1-clip; renal fibrosis; papilla; diagnostics; necrosis; non-coding RNAs; podocyte; Thy1.1 nephritis; KIT assay; oxidative stress; conditioned medium; C-reactive protein; pericyte; myofibroblast; Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-?B); endotoxemia; modifier gene; polymorphism; renal stem cells; kidney; polyploidization; Class IIa Histone Deacetylases (HDAC); 2k1c; molecular signaling; proximal tubule; arachidonic acid; empagliflozin; tubular injury; signaling cascade; signal transduction; inflammatory maker; niches; biomarkers; renal progenitors; type V collagen; cyclooxygenase; focal segmental glomerulosclerosis; inflammatory bowel disease (IBD); chronic kidney disease (CKD); allograft rejection; renovascular hypertension; genotype; molecular mechanisms; ROS; prediction; glomerular filtration barrier (GFB); alport syndrome; scattered tubular cells; long non-coding RNAs; renal inflammation; lysophosphatidic acid receptor; cAMP Regulatory Element Binding Protein (CREB); Farnesiferol B; differentiation; mesenchymal stromal cells; modified-MSCs; kidney transplantation; polyunsaturated fatty acids; apoptosis; type I collagen; diabetes mellitus; natural products; lipoxygenase; stem cell; T cell-mediated rejection; exosomes; renal injury; obese kidney fibrosis; kidney injury; cytotoxicity; mesenchymal stem cell; pigment nephropathy; mesodermal stem cell; ischemia-reperfusion; cytochrome P450; renal cell carcinoma; hematuria; B-cell attracting chemokine; microRNA; chemerin; glomerular basement membrane; glomerular damage; renal tubular cells; kidney proximal tubule; exosome; hypertension; diabetic nephropathy
• Online toegang: DOAB: download the publication; DOAB: description of the publication

Acute kidney injury (AKI) is still associated with high morbidity and mortality incidence rates, and also bears an elevated risk of subsequent chronic kidney disease. Although the kidney has a remarkable capacity for regeneration after injury and may recover completely depending on the type of renal...