Dysregulated transmethylation leading to hepatocellular carcinoma compromises redox homeostasis and glucose formation
Objective: The loss of liver glycine N-methyltransferase (GNMT) promotes liver steatosis and the transition to hepatocellular carcinoma (HCC). Previous work showed endogenous glucose production is reduced in GNMT-null mice with gluconeogenic precursors being used in alternative biosynthetic pathways...
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| Main Authors: | , , , , |
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| Format: | Book |
| Published: |
Elsevier,
2019-05-01T00:00:00Z.
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| Online Access: | Connect to this object online. |
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Internet
Connect to this object online.3rd Floor Main Library
| Call Number: |
A1234.567 |
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| Copy 1 | Available |