Smad3-Targeted Therapy Protects against Cisplatin-Induced AKI by Attenuating Programmed Cell Death and Inflammation via a NOX4-Dependent Mechanism
Background: Transforming growth factor-β (TGF-β)/Smad signaling is the central mediator in renal fibrosis, yet its functional role in acute kidney injury (AKI) is not fully understood. Recent evidence showed that TGF-β/Smad3 may be involved in the pathogenesis of AKI, but its functional role and mec...
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Main Authors: | , , , , , , , |
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פורמט: | ספר |
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Karger Publishers,
2021-02-01T00:00:00Z.
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Connect to this object online.3rd Floor Main Library
סימן המיקום: |
A1234.567 |
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עותק 1 | זמין |