The Effects of Ginsenoside Compound K Against Epilepsy by Enhancing the γ-Aminobutyric Acid Signaling Pathway

• Main Authors: Xiangchang Zeng (Author), Kai Hu (Author), Lulu Chen (Author), Luping Zhou (Author), Wei Luo (Author), Chaopeng Li (Author), Wenjing Zong (Author), Siyu Chen (Author), Qing Gao (Author), Guirong Zeng (Author), Dejian Jiang (Author), Xiaohui Li (Author), Honghao Zhou (Author), Dong-sheng Ouyang (Author)
• Format: Book
• Published: Frontiers Media S.A., 2018-09-01T00:00:00Z.
• Subjects: article
• Online Access: Connect to this object online.

The imbalance between the GABA-mediated inhibition and the glutamate-mediated excitation is the primary pathological mechanism of epilepsy. GABAergic and glutamatergic neurotransmission have become the most important targets for controlling epilepsy. Ginsenoside compound K (GCK) is a main metabolic...