Trypanosoma brucei TbIF1 inhibits the essential F1-ATPase in the infectious form of the parasite.
The mitochondrial (mt) FoF1-ATP synthase of the digenetic parasite, Trypanosoma brucei, generates ATP during the insect procyclic form (PF), but becomes a perpetual consumer of ATP in the mammalian bloodstream form (BF), which lacks a canonical respiratory chain. This unconventional dependence on Fo...
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| Main Authors: | , , |
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| Format: | Book |
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Public Library of Science (PLoS),
2017-04-01T00:00:00Z.
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| Online Access: | Connect to this object online. |
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Internet
Connect to this object online.3rd Floor Main Library
| Call Number: |
A1234.567 |
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| Copy 1 | Available |