DYNLT1 promotes mitochondrial metabolism to fuel breast cancer development by inhibiting ubiquitination degradation of VDAC1

Abstract Background Mitochondrial metabolism has been proposed as an attractive target for breast cancer therapy. The discovery of new mechanisms underlying mitochondrial dysfunction will facilitate the development of new metabolic inhibitors to improve the clinical treatment of breast cancer patien...

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Asıl Yazarlar: Ling Huang (Yazar), Bo Wei (Yazar), Yuran Zhao (Yazar), Xue Gong (Yazar), Liming Chen (Yazar)
Materyal Türü: Kitap
Baskı/Yayın Bilgisi: BMC, 2023-06-01T00:00:00Z.
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