Silibinin ameliorates hepatic lipid accumulation and oxidative stress in mice with non-alcoholic steatohepatitis by regulating CFLAR-JNK pathway

• المؤلفون الرئيسيون: Yayun Liu (مؤلف), Wei Xu (مؤلف), Ting Zhai (مؤلف), Jiaojiao You (مؤلف), Yong Chen (مؤلف)
• التنسيق: كتاب
• منشور في: Elsevier, 2019-07-01T00:00:00Z.
• الموضوعات: article
• الوصول للمادة أونلاين: Connect to this object online.

Non-alcoholic steatohepatitis (NASH) is a chronic metabolic syndrome and the CFLAR-JNK pathway can reverse the process of NASH. Although silibinin is used for the treatment of NASH in clinical, its effect on CFLAR-JNK pathway in NASH remains unclear. This study aimed to investigate the effect of sil...