SMYD3-PARP16 axis accelerates unfolded protein response and mediates neointima formation

• Main Authors: Fen Long (Author), Di Yang (Author), Jinghua Wang (Author), Qing Wang (Author), Ting Ni (Author), Gang Wei (Author), Yizhun Zhu (Author), Xinhua Liu (Author)
• Format: Book
• Published: Elsevier, 2021-05-01T00:00:00Z.
• Subjects: article
• Online Access: Connect to this object online.

Neointimal hyperplasia after vascular injury is a representative complication of restenosis. Endoplasmic reticulum (ER) stress-induced unfolded protein response (UPR) is involved in the pathogenesis of vascular intimal hyperplasia. PARP16, a member of the poly(ADP-ribose) polymerases family, is corr...