An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (A-T)
Background: ATM, the protein defective in the human genetic disorder, ataxia-telangiectasia (A-T) plays a central role in response to DNA double-strand breaks (DSBs) and in protecting the cell against oxidative stress. We showed that A-T cells are hypersensitive to metabolic stress which can be acco...
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Elsevier,
2021-12-01T00:00:00Z.
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