Contribution of Altered Endocannabinoid System to Overactive mTORC1 Signaling in Focal Cortical Dysplasia

• Asıl Yazarlar: Daniel García-Rincón (Yazar), Javier Díaz-Alonso (Yazar), Juan Paraíso-Luna (Yazar), Zaira Ortega (Yazar), José Aguareles (Yazar), Adán de Salas-Quiroga (Yazar), Cristina Jou (Yazar), Inmaculada de Prada (Yazar), Verónica Martínez-Cerdeño (Yazar), Eleonora Aronica (Yazar), Manuel Guzmán (Yazar), María Ángeles Pérez-Jiménez (Yazar), Ismael Galve-Roperh (Yazar)
• Materyal Türü: Kitap
• Baskı/Yayın Bilgisi: Frontiers Media S.A., 2019-01-01T00:00:00Z.
• Konular: article
• Online Erişim: Connect to this object online.

Alterations of the PI3K/Akt/mammalian target of rapamycin complex 1 (mTORC1) signaling pathway are causally involved in a subset of malformations of cortical development (MCDs) ranging from focal cortical dysplasia (FCD) to hemimegalencephaly and megalencephaly. These MCDs represent a frequent cause...