Xingnaojing ameliorates synaptic plasticity and memory deficits in an Aβ1-42 induced mouse model of Alzheimer's disease
The accumulation of insoluble amyloid β (Aβ) peptides is one of the pathological changes in Alzheimer's disease (AD), which induced synaptic plasticity impairment and excitatory amino acid toxicity associated with decreased memory function. Xingnaojing (XNJ), a well-known prescription in tradit...
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Main Authors: | , , , , , , , , , , , , |
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Format: | Book |
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Elsevier,
2020-08-01T00:00:00Z.
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A1234.567 |
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