Xingnaojing ameliorates synaptic plasticity and memory deficits in an Aβ1-42 induced mouse model of Alzheimer's disease

• Главные авторы: Yi Liu (Автор), Siyi Xu (Автор), Huijie Bian (Автор), Yi Qian (Автор), Huiya Li (Автор), Shu Shu (Автор), Jiang Chen (Автор), Xiang Cao (Автор), Yue Gu (Автор), Jiali Jin (Автор), Xi Zhang (Автор), Yun Xu (Автор), Xiaolei Zhu (Автор)
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• Опубликовано: Elsevier, 2020-08-01T00:00:00Z.
• Предметы: article
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The accumulation of insoluble amyloid β (Aβ) peptides is one of the pathological changes in Alzheimer's disease (AD), which induced synaptic plasticity impairment and excitatory amino acid toxicity associated with decreased memory function. Xingnaojing (XNJ), a well-known prescription in tradit...