Xingnaojing ameliorates synaptic plasticity and memory deficits in an Aβ1-42 induced mouse model of Alzheimer's disease

• Main Authors: Yi Liu (Author), Siyi Xu (Author), Huijie Bian (Author), Yi Qian (Author), Huiya Li (Author), Shu Shu (Author), Jiang Chen (Author), Xiang Cao (Author), Yue Gu (Author), Jiali Jin (Author), Xi Zhang (Author), Yun Xu (Author), Xiaolei Zhu (Author)
• Format: Book
• Published: Elsevier, 2020-08-01T00:00:00Z.
• Subjects: article
• Online Access: Connect to this object online.

The accumulation of insoluble amyloid β (Aβ) peptides is one of the pathological changes in Alzheimer's disease (AD), which induced synaptic plasticity impairment and excitatory amino acid toxicity associated with decreased memory function. Xingnaojing (XNJ), a well-known prescription in tradit...