PAQR9 regulates hepatic ketogenesis and fatty acid oxidation during fasting by modulating protein stability of PPARα
Background: The cycle of feeding and fasting is fundamental to life and closely coordinated with changes of metabolic programs. During extended starvation, ketogenesis coupled with fatty acid oxidation in the liver supplies ketone bodies to extrahepatic tissues as the major form of fuel. In this stu...
Saved in:
Main Authors: | Yijun Lin (Author), Lingling Chen (Author), Xue You (Author), Zixuan Li (Author), Chenchen Li (Author), Yan Chen (Author) |
---|---|
Format: | Book |
Published: |
Elsevier,
2021-11-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Adipose tissue lipolysis is regulated by PAQR11 via altering protein stability of phosphodiesterase 4D
by: Meiqin Huang, et al.
Published: (2021) -
Hepatic ketogenesis is not required for starvation adaptation in mice
by: Kyle Feola, et al.
Published: (2024) -
Alleviation of Toxicity Caused by Overactivation of Pparα through Pparα-Inducible miR-181a2
by: Yanjie Cheng, et al.
Published: (2017) -
Crosstalk interactions between transcription factors ERRα and PPARα assist PPARα-mediated gene expression
by: Sofie J. Desmet, et al.
Published: (2024) -
Hmgcs2-mediated ketogenesis modulates high-fat diet-induced hepatosteatosis
by: Shaza Asif, et al.
Published: (2022)