Pathophysiological Consequences of KATP Channel Overactivity and Pharmacological Response to Glibenclamide in Skeletal Muscle of a Murine Model of Cantù Syndrome

Cantù syndrome (CS) arises from mutations in ABCC9 and KCNJ8 genes that lead to gain of function (GOF) of ATP-sensitive potassium (KATP) channels containing SUR2A and Kir6.1 subunits, respectively, of KATP channels. Pathological consequences of CS have been reported for cardiac and smooth muscle cel...

Full description

Saved in:
Bibliographic Details
Main Authors: Rosa Scala (Author), Fatima Maqoud (Author), Nicola Zizzo (Author), Antonietta Mele (Author), Giulia Maria Camerino (Author), Francesco Alfredo Zito (Author), Girolamo Ranieri (Author), Conor McClenaghan (Author), Theresa M. Harter (Author), Colin G. Nichols (Author), Domenico Tricarico (Author)
Format: Book
Published: Frontiers Media S.A., 2020-11-01T00:00:00Z.
Subjects:
Online Access:Connect to this object online.
Tags: Add Tag
No Tags, Be the first to tag this record!

Internet

Connect to this object online.

3rd Floor Main Library

Holdings details from 3rd Floor Main Library
Call Number: A1234.567
Copy 1 Available