PHF5A promotes colorectal cancerprogression by alternative splicing of TEAD2

• Egile Nagusiak: Yue Chang (Egilea), Yulu Zhao (Egilea), Liya Wang (Egilea), Meijuan Wu (Egilea), Chenglong He (Egilea), Mengxi Huang (Egilea), Zengjie Lei (Egilea), Jiahe Yang (Egilea), Siqi Han (Egilea), Bibo Wang (Egilea), Yanyan Chen (Egilea), Chao Liu (Egilea), Hongju Yu (Egilea), Lijun Xue (Egilea), Jian Geng (Egilea), Yanan Chen (Egilea), Tingting Dai (Egilea), Lili Ren (Egilea), Qian Wang (Egilea), Xiaobei Liu (Egilea), Xiaoyuan Chu (Egilea), Cheng Chen (Egilea)
• Formatua: Liburua
• Argitaratua: Elsevier, 2021-12-01T00:00:00Z.
• Gaiak: article
• Sarrera elektronikoa: Connect to this object online.

Dysregulated alternative splicing (AS) plays critical roles in driving cancer progression, and the underlying mechanisms remain largely unknown. Here, we demonstrated that PHF5A, a component of U2 small nuclear ribonucleoproteins, was frequently upregulated in colorectal cancer (CRC) samples and ass...