Conditional deletion of HDAC4 from collagen type 2α1-expressing cells increases angiogenesis in vivo
Abstract Background HDAC4 is a key regulator of chondrocyte hypertrophy and skeletal development, but it is not clear whether the increase in vascular invasion at growth plates is related to HDAC4 expression. To determine it, we investigated the relationship between HDAC4 and angiogenesis in both in...
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Format: | Book |
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BMC,
2020-05-01T00:00:00Z.
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A1234.567 |
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