A deletion of <it>FGFR2 </it>creating a chimeric IIIb/IIIc exon in a child with Apert syndrome
<p>Abstract</p> <p>Background</p> <p>Signalling by fibroblast growth factor receptor type 2 (FGFR2) normally involves a tissue-specific alternative splice choice between two exons (IIIb and IIIc), which generates two receptor isoforms (FGFR2b and FGFR2c respectively) wi...
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Main Authors: | , , , |
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Formato: | Libro |
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BMC,
2011-09-01T00:00:00Z.
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Acceso en liña: | Connect to this object online. |
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Número de Clasificación: |
A1234.567 |
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Copia 1 | Dispoñible |