Cardiomyocytes, cardiac endothelial cells and fibroblasts contribute to anthracycline-induced cardiac injury through RAS-homologous small GTPases RAC1 and CDC42
The clinical use of the DNA damaging anticancer drug doxorubicin (DOX) is limited by irreversible cardiotoxicity, which depends on the cumulative dose. The RAS-homologous (RHO) small GTPase RAC1 contributes to DOX-induced DNA damage formation and cardiotoxicity. However, the pathophysiological relev...
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| Main Authors: | , , , , |
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| Format: | Book |
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Elsevier,
2024-05-01T00:00:00Z.
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| Call Number: |
A1234.567 |
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| Copy 1 | Available |