RIPK3 promotes islet amyloid-induced β-cell loss and glucose intolerance in a humanized mouse model of type 2 diabetes

Objective: Aggregation of human islet amyloid polypeptide (hIAPP), a β-cell secretory product, leads to islet amyloid deposition, islet inflammation and β-cell loss in type 2 diabetes (T2D), but the mechanisms that underlie this process are incompletely understood. Receptor interacting protein kinas...

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主要な著者: Noyonika Mukherjee (著者), Christopher J. Contreras (著者), Li Lin (著者), Kaitlyn A. Colglazier (著者), Egan G. Mather (著者), Michael A. Kalwat (著者), Nathalie Esser (著者), Steven E. Kahn (著者), Andrew T. Templin (著者)
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出版事項: Elsevier, 2024-02-01T00:00:00Z.
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