Gut bacterial metabolite Urolithin A inhibits myocardial fibrosis through activation of Nrf2 pathway in vitro and in vivo
Abstract Background Myocardial fibrosis after myocardial infarction (MI) is one of the leading causes of cardiovascular diseases. Cardiac fibroblasts (CFs) are activated and promoted by MI to undergo myofibroblast transformation (CMT). Urolithin A (UA) is an active and effective gut metabolite deriv...
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| Main Authors: | , , , , , |
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| Format: | Book |
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BMC,
2022-02-01T00:00:00Z.
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Internet
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| Call Number: |
A1234.567 |
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