Acyl CoA synthetase 5 (ACSL5) ablation in mice increases energy expenditure and insulin sensitivity and delays fat absorption

Acyl CoA synthetase 5 (ACSL5) ablation in mice increases energy expenditure and insulin sensitivity and delays fat absorption

Objective: The family of acyl-CoA synthetase enzymes (ACSL) activates fatty acids within cells to generate long chain fatty acyl CoA (FACoA). The differing metabolic fates of FACoAs such as incorporation into neutral lipids, phospholipids, and oxidation pathways are differentially regulated by the A...

Full description

Saved in:
Bibliographic Details
Main Authors: Thomas A. Bowman (Author), Kayleigh R. O'Keeffe (Author), Theresa D'Aquila (Author), Qing Wu Yan (Author), John D. Griffin (Author), Elizabeth A. Killion (Author), Deanna M. Salter (Author), Douglas G. Mashek (Author), Kimberly K. Buhman (Author), Andrew S. Greenberg (Author)
Format: Book
Published: Elsevier, 2016-03-01T00:00:00Z.
Subjects:
article
Online Access:Connect to this object online.
Tags: Add Tag
No Tags, Be the first to tag this record!

Internet

Connect to this object online.

3rd Floor Main Library

Holdings details from 3rd Floor Main Library
Call Number: A1234.567
Copy 1 Available

Similar Items