Reverse Electron Transport at Mitochondrial Complex I in Ischemic Stroke, Aging, and Age-Related Diseases
Stroke is one of the leading causes of morbidity and mortality worldwide. A main cause of brain damage by stroke is ischemia-reperfusion (IR) injury due to the increased production of reactive oxygen species (ROS) and energy failure caused by changes in mitochondrial metabolism. Ischemia causes a bu...
Saved in:
Main Authors: | Vishal Chavda (Author), Bingwei Lu (Author) |
---|---|
Format: | Book |
Published: |
MDPI AG,
2023-04-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Physiologic Implications of Reactive Oxygen Species Production by Mitochondrial Complex I Reverse Electron Transport
by: John O. Onukwufor, et al.
Published: (2019) -
Ischemic Stroke and SARS-CoV-2 Infection: The Bidirectional Pathology and Risk Morbidities
by: Vishal Chavda, et al.
Published: (2022) -
Pramipexole prevents ischemic cell death via mitochondrial pathways in ischemic stroke
by: Syed Suhail Andrabi, et al.
Published: (2019) -
Mitochondrial Complex I, a Possible Sensible Site of cAMP Pathway in Aging
by: Anna Signorile, et al.
Published: (2023) -
New TIPARP inhibitor rescues mitochondrial function and brain injury in ischemic stroke
by: Yang Cai, et al.
Published: (2024)