Cigarette smoke extract-mediated FABP4 upregulation suppresses viability and induces apoptosis, inflammation and oxidative stress of bronchial epithelial cells by activating p38 MAPK/MK2 signaling pathway
Abstract Background Long-term inhalation of cigarette smoke is considered to be one of the main causes of bronchial epithelioid cell damage, but its underlying mechanism has to be further clarified. Methods Gene expression at mRNA level and protein levels were detected by qRT-PCR and western blot an...
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| Format: | Book |
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BMC,
2022-06-01T00:00:00Z.
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