New Challenges Resulting From the Loss of Function of Nav1.4 in Neuromuscular Diseases
The voltage-gated sodium channel Nav1.4 is a major actor in the excitability of skeletal myofibers, driving the muscle force in response to nerve stimulation. Supporting further this key role, mutations in SCN4A, the gene encoding the pore-forming α subunit of Nav1.4, are responsible for a clinical...
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| Main Authors: | , |
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| Format: | Book |
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Frontiers Media S.A.,
2021-10-01T00:00:00Z.
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A1234.567 |
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