Hepatic GRK2 is dispensable for glucose homeostasis and other key metabolic parameters in mice
Objective: G-protein-coupled receptor (GPCR) kinases (GRKs) abrogate GPCR signaling by promoting receptor desensitization and internalization. Accumulating evidence suggests that GRK2 represents an important regulator of GPCR-mediated effects on systemic glucose metabolism, obesity, and insulin resi...
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| Main Authors: | , , , , |
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| Format: | Book |
| Published: |
Elsevier,
2024-01-01T00:00:00Z.
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| Subjects: | |
| Online Access: | Connect to this object online. |
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