Allicin disrupts cardiac Cav1.2 channels via trafficking

• Asıl Yazarlar: Dan Han (Yazar), Lingping Xu (Yazar), Peng Liu (Yazar), Yingying Liu (Yazar), Chaofeng Sun (Yazar), Yanrong Yin (Yazar)
• Materyal Türü: Kitap
• Baskı/Yayın Bilgisi: Taylor & Francis Group, 2019-01-01T00:00:00Z.
• Konular: article
• Online Erişim: Connect to this object online.

Context: Allicin is a potential antiarrhythmic agent. The antiarrhythmic properties of allicin rely on its blockade of various cardiac ion channels. The l-type calcium (Cav1.2) channel provides a pivotal substrate for cardiac electrophysiologic activities. The mechanism of allicin on Cav1.2 remains...