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Overexpression of PEP-19 Suppresses Angiotensin II-Induced Cardiomyocyte Hypertrophy

Abstract.: The precise molecular mechanisms leading to disturbance of Ca2+/calmodulin-dependent intracellular signalling in cardiac hypertrophy remains unclear. As an endogenous calmodulin regulator protein, the pathophysiology role of PEP-19 during cardiac hypertrophy was investigated in the presen...

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Main Authors: Yang-yang Xie (Author), Meng-meng Sun (Author), Xue-fang Lou (Author), Chen Zhang (Author), Feng Han (Author), Bo-ya Zhang (Author), Ping Wang (Author), Ying-mei Lu (Author)
Format: Book
Published: Elsevier, 2014-01-01T00:00:00Z.
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