Overexpression of PEP-19 Suppresses Angiotensin II-Induced Cardiomyocyte Hypertrophy
Abstract.: The precise molecular mechanisms leading to disturbance of Ca2+/calmodulin-dependent intracellular signalling in cardiac hypertrophy remains unclear. As an endogenous calmodulin regulator protein, the pathophysiology role of PEP-19 during cardiac hypertrophy was investigated in the presen...
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Elsevier,
2014-01-01T00:00:00Z.
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Internet
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A1234.567 |
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Kopie 1 | Beschikbaar |