Mouse models for preeclampsia: disruption of redox-regulated signaling
<p>Abstract</p> <p>The concept that oxidative stress contributes to the development of human preeclampsia has never been tested in genetically-defined animal models. Homozygous deletion of catechol-O-methyl transferase (Comt-/-) in pregnant mice leads to human preeclampsia-like sym...
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| Main Authors: | , , |
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| Format: | Book |
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BMC,
2009-01-01T00:00:00Z.
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| Call Number: |
A1234.567 |
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