Characterization of an N-terminal Nav1.5 channel variant - a potential risk factor for arrhythmias and sudden death?
Abstract Background Alterations in the SCN5A gene encoding the cardiac sodium channel Nav1.5 have been linked to a number of arrhythmia syndromes and diseases including long-QT syndrome (LQTS), Brugada syndrome (BrS) and dilative cardiomyopathy (DCM), which may predispose to fatal arrhythmias and su...
Saved in:
Main Authors: | Stefanie Scheiper-Welling (Author), Paolo Zuccolini (Author), Oliver Rauh (Author), Britt-Maria Beckmann (Author), Christof Geisen (Author), Anna Moroni (Author), Gerhard Thiel (Author), Silke Kauferstein (Author) |
---|---|
Format: | Book |
Published: |
BMC,
2020-11-01T00:00:00Z.
|
Subjects: | |
Online Access: | Connect to this object online. |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Characterization of a novel KCNJ2 sequence variant detected in Andersen-Tawil syndrome patients
by: Stefanie Scheiper, et al.
Published: (2017) -
Left-shifted Nav channels in injured bilayer: primary targets for neuroprotective Nav antagonists?
by: Catherine E Morris, et al.
Published: (2012) -
Nav channels in cancers: Non-classical roles
by: Hengrui Liu
Published: (2020) -
Acidosis differentially modulates inactivation in NaV1.2, NaV1.4, and NaV1.5 channels
by: Yury Y. Vilin, et al.
Published: (2012) -
Carvacrol inhibits the neuronal voltage-gated sodium channels Nav1.2, Nav1.6, Nav1.3, Nav1.7, and Nav1.8 expressed in Xenopus oocytes with different potencies
by: Takafumi Horishita, et al.
Published: (2020)