Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na<sub>V</sub>1.7 produce distinct pain disorders

Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na<sub>V</sub>1.7 produce distinct pain disorders

<p>Abstract</p> <p>Background</p> <p>Two groups of gain-of-function mutations in sodium channel Na<sub>V</sub>1.7, which are expressed in dorsal root ganglion (DRG) neurons, produce two clinically-distinct pain syndromes - inherited erythromelalgia (IEM) and...

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Main Authors: Tyrrell Lynda (Author), Dib-Hajj Sulayman D (Author), Cheng Xiaoyang (Author), Wright Dowain A (Author), Fischer Tanya Z (Author), Waxman Stephen G (Author)
Format: Book
Published: SAGE Publishing, 2010-04-01T00:00:00Z.
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3rd Floor Main Library

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